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Lactylation-driven METTL3 regulates wound healing by enhancing m6A/HNRNPA2B1/DNMT1 signaling in keratinocytes 认领 引用
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作者 Xue-ting Hu Lu-min Sui +6 位作者 Cheng-xiu Pu Luo-quan Ao Mu Yuan Li Deng Qing Zhao Xiao-feng Wu Xiang Xu 《Genes & Diseases》 SCIE CSCD 2026年第3期86-89,共4页
Chronic non-healing wounds,such as diabetic foot ulcers,represent a clinical challenge with an increasing incidence.1 A deeper understanding of the molecular mechanisms underlying wound healing is of great significanc... Chronic non-healing wounds,such as diabetic foot ulcers,represent a clinical challenge with an increasing incidence.1 A deeper understanding of the molecular mechanisms underlying wound healing is of great significance.RNA m6A modification is a widespread and important epigenetic regulatory mechanism,2 but its role in wound healing remains unclear.In this study,we found that METTL3,an RNA m6A methyltransferase,is a positive regulator of wound healing and that its low expression is closely related to chronic diabetic wounds. 展开更多
关键词 wound healing rna m methyltransferaseis chronic d molecular mechanisms underlying m modification epigenetic regulatory mechanism diabetic foot ulcersrepresent lactylation driven
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